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Switching on Plant Innate Immunity Signaling Systems: Bioengineering and Molecular Manipulation of PAMP-PIMP-PRR Signaling Complex 2016 ed. [Hardback]

  • Formāts: Hardback, 358 pages, height x width: 235x155 mm, weight: 6801 g, 1 Illustrations, color; 91 Illustrations, black and white; XIII, 358 p. 92 illus., 1 illus. in color., 1 Hardback
  • Sērija : Signaling and Communication in Plants
  • Izdošanas datums: 29-Jan-2016
  • Izdevniecība: Springer International Publishing AG
  • ISBN-10: 3319261169
  • ISBN-13: 9783319261164
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Switching on Plant Innate Immunity Signaling Systems: Bioengineering and Molecular Manipulation of PAMP-PIMP-PRR Signaling Complex 2016 ed.Palielināt
  • Formāts: Hardback, 358 pages, height x width: 235x155 mm, weight: 6801 g, 1 Illustrations, color; 91 Illustrations, black and white; XIII, 358 p. 92 illus., 1 illus. in color., 1 Hardback
  • Sērija : Signaling and Communication in Plants
  • Izdošanas datums: 29-Jan-2016
  • Izdevniecība: Springer International Publishing AG
  • ISBN-10: 3319261169
  • ISBN-13: 9783319261164
Citas grāmatas par šo tēmu:
Permanent link: https://www.kriso.lv/db/9783319261164.html
Keywords:
This book presents the ways and means to switch on plant immune signaling systems using PAMP-PIMP-PRR signaling complex for crop disease management. It also describes bioengineering approaches to develop transgenic plants expressing enhanced disease resistance using genes encoding PAMPs, PRRs and transcription factors and genes involved in generation of PIMPs/HAMPs. It also discusses recent commercial development of PAMP products to switch on plant innate immunity for crop disease management. These unique approaches have been described with more than 100 figures and illustrations and these would make this book attractive for researchers and students to buy this book.
1 Introduction 1(26)
1.1 Plant Innate Immunity Is a Sleeping Giant to Fight against Pathogens
2(1)
1.2 Potential Signals to Switch on Plant Immune System
3(1)
1.3 Pathogens Possess Weapons to Switch-Off Plant Immune Systems
4(1)
1.4 Bioengineering and Molecular Manipulation Technologies to Switch on the Sleeping Quiescent Plant Immune System to Win the War against Pathogens
5(4)
1.5 Switching on Plant Innate Immunity Using PAMP-PIMP-PRR-Transcription Factor Is the Most Potential Biotechnological Approach for Management of Crop Diseases
9(1)
References
10(17)
2 Role of Plant Immune Signals and Signaling Systems in Plant Pathogenesis 27(64)
2.1 Susceptibility and Disease Resistance Are Two Sides of the Same Coin Modulated by Plant Immune System Signals and Signaling Systems
29(1)
2.2 Signals and Signaling Systems Involved in Triggering Immune Responses
30(7)
2.2.1 PAMP-PRR Signaling Complex in Triggering Immune Responses
30(2)
2.2.2 PAMPs Activate Cat Signaling Systems
32(1)
2.2.3 PAMPs Activate G-Protein Signaling
32(1)
2.2.4 PAMPs Activate ROS Signaling System
33(1)
2.2.5 PAMPs Activate NO Signaling System
33(1)
2.2.6 PAMPs Activate Mitogen-Activated Protein Kinase Signaling System
34(1)
2.2.7 PAMPs Activate Salicylic Acid Signaling System
34(1)
2.2.8 PAMPs Activate Jasmonate Signaling System
35(1)
2.2.9 PAMPs Activate Ethylene Signaling System
36(1)
2.2.10 PAMPs Trigger ABA Signaling System
36(1)
2.2.11 PAMPs Trigger Expression of Transcription Factors
37(1)
2.3 Reduced Activity of PAMPs May Facilitate the Virulent Pathogens to Cause Disease
37(2)
2.3.1 Pathogen May Modify Its PAMP Structure during Its Pathogenesis to Reduce Its Elicitor Activity
37(1)
2.3.2 Virulent Pathogen May Contain Inefficient PAMP and Trigger Subdued Defense Responses Favoring Disease Development
38(1)
2.4 Pathogen-Secreted Effectors Suppress PAMP-Triggered Plant Immune Responses
39(9)
2.4.1 Pathogen-Secreted Effector Molecules
39(1)
2.4.2 Effectors Suppress PAMP-Triggered Plant Immunity
39(2)
2.4.3 Effectors May Disrupt Binding of PAMP with PRR in PAMP-PRR Signaling Complex to Impede PAMP-Triggered Plant Immunity
41(1)
2.4.4 Effectors May Promote Ubiquitin-Proteasome-Mediated Degradation of PRRs to Impede PAMP-Triggered Plant Immunity
42(1)
2.4.5 Effectors May Target the Kinase Domains of PRR and Inhibit the PRR Receptor Kinase Activity to Block PAMP-Triggered Immunity
42(1)
2.4.6 Effectors May Inhibit Autophosphorylation of PRRs
43(1)
2.4.7 Effectors May Bind With the PRR Signal Amplifier BAK1 and Block the Function of PAMP-PRR Signaling Complex
43(1)
2.4.8 Effectors May Target the Receptor-Like Cytoplasmic Kinases BIK1 and PBL1
44(1)
2.4.9 Effector Suppresses MAPK Signaling to Promote Disease Development
45(2)
2.4.10 Effectors May Suppress SA Signaling System to Facilitate Pathogenesis
47(1)
2.4.11 Effector May Subvert Ubiquitin-Proteasome System to Suppress PAMP-Triggered Immunity
47(1)
2.5 Host Plants May Manipulate the Defense Signaling Systems to Suppress the Disease Development
48(1)
2.6 Specificity of Plant Hormone Signaling Systems in Conferring Resistance against Various Pathogens
48(5)
2.7 Plant Hormone Signaling Systems May Also Induce Susceptibility against Pathogens
53(2)
2.8 Pathogens May Hijack Specific Signaling Pathways to Cause Disease
55(6)
2.8.1 Pathogens May Hijack ABA Signaling Pathway to Cause Disease
55(2)
2.8.2 Pathogens May Hijack ET Signaling System to Cause Disease
57(1)
2.8.3 Pathogens May Hijack JA Signaling System to Cause Disease
58(1)
2.8.4 Pathogen May Hijack Auxin Metabolism to Cause Disease
59(1)
2.8.5 Pathogen Hijacks Brassinosteroid Signaling Machinery to Cause Disease
60(1)
2.9 Pathogens May Suppress Specific Signaling System to Promote Disease Development
61(6)
2.9.1 Pathogens May Suppress SA Signaling System to Promote Disease Development
61(3)
2.9.2 Pathogens May Suppress JA Signaling System to Promote Disease Development
64(1)
2.9.3 Pathogen May Suppress ABA Signaling System to Promote Pathogenesis
64(1)
2.9.4 Pathogens May Suppress GA Signaling Pathway to Cause Disease
65(1)
2.9.5 Pathogens May Suppress ROS Signaling System to Promote Disease Development
65(2)
2.9.6 Viral Pathogens May Inhibit Ubiquitin-Proteasome System to Induce Disease Development
67(1)
References
67(24)
3 Switching on Plant Immune Signaling Systems Using Microbe-Associated Molecular Patterns 91(100)
3.1 PAMP-Triggered Immunity
93(5)
3.1.1 PAMPs Detected in Bacterial, Fungal, Oomycete, and Viral Pathogens
93(4)
3.1.2 Variability in Structure and Function of PAMPs
97(1)
3.2 Harpin PAMPs as Molecular Tools to Manipulate PAMP-Triggered Immunity
98(18)
3.2.1 Harpins Acting as PAMPs
98(3)
3.2.2 Harpin-Induced Plant Immune Signal Transduction Systems
101(6)
3.2.3 Harpin-Induced Defense Response Genes
107(2)
3.2.4 Development of Harpin Formulations for Management of Crop Diseases
109(1)
3.2.5 Foliar Application of Harpin Induces Plant Immune Responses against Wide Range of Pathogens
109(2)
3.2.6 Harpin Treatment Triggers SA-Dependent Systemic Acquired Resistance
111(2)
3.2.7 Time of Application of Harpin Determines Its Efficacy in Induction of Defense Response
113(1)
3.2.8 Amount of Harpin Determines Its Efficacy in Inducing Disease Resistance
114(1)
3.2.9 Harpin Increases Crop Growth and Crop Yield
115(1)
3.2.10 Foliar Spray Application of Bacillus thuringiensis Expressing Harpin Gene
115(1)
3.3 Engineering Harpin Gene to Develop Disease Resistant Plants
116(6)
3.3.1 Transgenic Plants Expressing the Bacterial Harpin Gene Show Enhanced Resistance against the Bacterial Pathogen
116(1)
3.3.2 Harpin Gene from a Bacterial Pathogen Triggers Defense Responses against Viral, Fungal, Oomycete and Also Bacterial Pathogens in Different Host Plants
117(3)
3.3.3 Variation in Levels of Harpin Gene Expression Resulting in Variation in Levels of Expression of Disease Resistance
120(2)
3.3.4 Growth and Yield Potential of Transgenic Plants Expressing Bacterial Harpin Gene
122(1)
3.4 Molecular Manipulation of Plant Innate Immune Signaling Systems Using Flagellin
122(6)
3.4.1 Activation of Plant Immune Signaling System by Flg22
122(5)
3.4.2 Flg22 Triggers Host Defense Responses
127(1)
3.4.3 Foliar Application of Flg22 Induces Disease Resistance
127(1)
3.4.4 Genetic Engineering to Develop Disease Resistant Plants Using Flagellin
128(1)
3.5 Molecular Manipulation of Plant Immune Systems Using the PAMP Elicitins
128(16)
3.5.1 Oomycetes-Secreted Elicitins
128(1)
3.5.2 Elicitin-Induced Early Plant Immune Signaling Events
129(10)
3.5.3 Induction of Salicylic Acid Biosynthesis and SA-Dependent Signaling Pathway by Elicitins
139(1)
3.5.4 Elicitin-Induced Jasmonic Acid Biosynthesis and JA-dependent Signaling Pathway
139(1)
3.5.5 Induction of Ethylene Biosynthesis and Ethylene-Dependent Signaling Pathway
140(1)
3.5.6 Elicitin-Induced Defense Responses
140(1)
3.5.7 Elicitins Trigger Systemic Acquired Resistance
141(1)
3.5.8 Management of Crop Diseases Using Elicitin
141(1)
3.5.9 Genetic Engineering to Develop Disease Resistant Plants Using Elicitin Gene
142(2)
3.6 Manipulation of Plant Immune System Using Chitosan
144(9)
3.6.1 Induction of Plant Defense Signaling Systems by Chitosan
144(1)
3.6.2 Induction of Host Defense Responses by Chitosan
144(4)
3.6.3 Chitosan Induces Resistance against Wide-Range of Pathogens
148(5)
3.7 Manipulation of Plant Immune System Using Cerebrosides
153(1)
3.8 Manipulation of Plant Immune System Using CfliNNI1 Elicitor
154(1)
3.9 Bioengineering FsphDNase Elicitor Gene to Trigger Plant Immune Responses against Wide Range of Pathogens
154(1)
3.10 Engineering the Elicitor-Encoding pemG1 Gene for Crop Disease Management
155(1)
3.11 Manipulation of Plant Immune System Using the MAMP Rhamnolipids
156(2)
3.11.1 Activation of Plant Immune Signaling System
156(2)
3.11.2 Potential of the MAMP Rhamnolipids for Management of Crop Diseases
158(1)
3.12 Manipulation of Plant Immune System Using the Proteinaceous Elicitor Sml Derived from Trichoderma virens
158(1)
3.13 Manipulation of Plant Immune Responses Using Yeast-Derived Elicitors
159(1)
References
160(31)
4 Switching on Plant Immune Signaling Systems Using Pathogen-Induced Molecular Patterns/Host-Associated Molecular Patterns 191(38)
4.1 Pathogen-Induced Molecular Patterns (PIMPs)/Host-Associated Molecular Patterns (HAMPs)
192(2)
4.2 Oligogalacturonides Switch on Plant Innate Immunity
194(4)
4.3 OGAs with Different Degrees of Polymerization Differ in Triggering Defense Responses
198(1)
4.4 Degree of Methyl Esterification of OGAs Modulates the Elicitor Activity of OGAs
199(2)
4.5 Ability of OGAs to Trigger Defense Responses May Depend on Their Level of Acetylation
201(1)
4.6 Engineering Pectin Methyl Esterase Genes to Develop Disease Resistant Plants
202(2)
4.7 Bioengineering Pectin Methyl Esterase Inhibitor Protein for Plant Disease Management
204(1)
4.8 Engineering PG Gene to Develop Disease Resistant Plants
204(1)
4.9 Engineering PGIP Gene to Develop Disease-Resistant Plants
205(3)
4.10 Manipulation of Oligogalacturonides by Salicylic Acid (SA) Analog to Induce Resistance against Pathogens
208(2)
4.11 Switching on Plant Immune Signaling Systems Using Plant Elicitor Peptides (Peps) for Disease Management
210(5)
4.11.1 Plant Elicitor Peptides
210(1)
4.11.2 Peps - Triggered Immune Signaling Systems
210(3)
4.11.3 Management of Crop Diseases Using Pep Proteins
213(1)
4.11.4 Engineering PROPEP Genes for Disease Management
214(1)
4.11.5 Engineering prePIP Genes to Amplify Immunity Induced by the PEP1 for Disease Management
214(1)
4.12 Switching on Plant Immune Signaling Systems Using Systemin for Disease Management
215(3)
4.12.1 Systemin
215(1)
4.12.2 Systemin-Triggered Immune Signaling Systems
216(1)
4.12.3 Engineering Prosystemin Gene to Develop Disease-Resistant Plants
217(1)
References
218(11)
5 Switching on Plant Immune Signaling Systems Using Pattern Recognition Receptor Complex 229(26)
5.1 Pattern Recognition Receptors (PRRs)
230(5)
5.2 Importance of PRRs in Triggering Defense Responses against Pathogens
235(2)
5.3 Engineering PRRs for Disease Management
237(4)
5.3.1 Engineering the PRR EFR for Crop Disease Management
237(2)
5.3.2 Engineering the PRR FLS2 for Plant Disease Management
239(1)
5.3.3 Engineering the PRR XA21 for Crop Disease Management
239(1)
5.3.4 Engineering WAK1 Receptors for Crop Disease Management
240(1)
5.4 PRR-Interacting Protein Complexes
241(3)
5.5 Engineering PRR-Interacting Protein Complexes for Crop Disease Management
244(1)
5.5.1 Engineering SOBIR1 Gene Encoding Receptor-Like Kinase Interacting with PRRs for Inducing Disease Resistance
244(1)
5.5.2 Engineering the PRR-Interacting ERECTA Gene for Disease Management
245(1)
References
245(10)
6 Molecular Manipulation of Transcription Factors, the Master Regulators of PAMP-Triggered Signaling Systems 255
6.1 Transcription Factors as 'Master Switches' Regulating Expression of Defense Genes in Plant Immune Signaling Systems
256(2)
6.2 PAMPs and PIMPs/HAMPs Trigger Expression of Transcription Factors
258(2)
6.3 Role of Transcription Factors in Regulation of Ca2+ Signaling System
260(4)
6.4 ROS-Regulated Expression of Transcription Factors
264(2)
6.5 MAPKs-Modulated Phosphorylation of Transcription Factors in Activation of Plant Immune Responses
266(2)
6.6 Transcription Factors Regulating Salicylate Signaling in Plant Innate Immune System
268(6)
6.6.1 Transcription Factors Triggering SA Biosynthesis
268(2)
6.6.2 SA Induces Enhanced Expression of Transcription Factors to Activate Transcription of Defense Genes
270(2)
6.6.3 Transcription Factors May Regulate SA-Mediated Plant Immune Signaling Systems
272(2)
6.7 Transcription Factors Regulating Jasmonate Signaling System in Plant Innate Immunity
274(3)
6.7.1 Transcription Factors Triggering JA Biosynthesis
274(2)
6.7.2 JA Induces Enhanced Expression of Transcription Factors
276(1)
6.7.3 Transcription Factors Triggering Expression of JA-Responsive Defense genes
276(1)
6.8 Transcription Factors Regulating Ethylene Signaling System in Plant Innate Immunity
277(1)
6.9 Transcription Factors May Trigger "Priming" of Defense Responses
278(3)
6.9.1 What Is Priming?
278(1)
6.9.2 Histone Modifications in Chromatin Structure May Be Involved in the Priming Process
278(1)
6.9.3 Priming in Systemic Acquired Resistance
279(1)
6.9.4 Plants May Inherit the Priming Phenomenon to Next-Generation SAR
279(1)
6.9.5 Priming of Transcription Factors in Plant Defense System
280(1)
6.10 Bioengineering WRKY Transcription Factors for Rice Disease Management
281(12)
6.10.1 WRKY Transcription Factors Regulating Plant Immune Responses
281(2)
6.10.2 Engineering OsWRKY13 Gene
283(3)
6.10.3 Engineering OsWRKY22 Gene
286(1)
6.10.4 Engineering OsWRKY30 Gene
286(1)
6.10.5 Engineering OsWRKY31 Gene
287(1)
6.10.6 Engineering OsWRKY42 Gene
287(2)
6.10.7 Engineering OsWRKY45 Gene
289(1)
6.10.8 Engineering OsWRKY47 Gene
290(1)
6.10.9 Engineering OsWRKY53 Gene
290(1)
6.10.10 Engineering OsWRKY71 Gene
291(1)
6.10.11 Engineering OsWRKY89 Gene
292(1)
6.11 Bioengineering WRKY Transcription Factors for Wheat Disease Management
293(1)
6.12 Bioengineering WRKY Transcription Factors for Tobacco Disease Management
293(6)
6.12.1 Engineering VvWRKY1 Gene from Vitis vinifera
293(2)
6.12.2 Engineering VvWRKY2 Gene from Vitis vinifera
295(1)
6.12.3 Engineering VpWRKY3 Gene from Chinese Wild Grapevine
295(1)
6.12.4 Engineering MdWRKY1 Gene from Apple
296(1)
6.12.5 Engineering GhWRKY15 Gene from Cotton
296(2)
6.12.6 Engineering GhWRKY39-1 and GhWRKY39 Genes from Cotton
298(1)
6.12.7 Engineering GhWRKY44 Gene from Cotton
298(1)
6.12.8 Engineering CaWRKY27 Gene from Capsicum annuum
298(1)
6.13 Bioengineering WRKY Transcription Factors for Management of Grapevine Diseases
299(1)
6.14 Search for Arabidopsis Transcription Factor Genes for Using as Tools for Engineering Disease-Resistant Plants
299(5)
6.14.1 WRKY33 Transcription Factor
299(2)
6.14.2 WRKY70 Transcription Factor
301(1)
6.14.3 WRKY18 Transcription Factor
302(1)
6.14.4 WRKY29 Transcription Factor
303(1)
6.14.5 WRKY7 Transcription Factor
303(1)
6.14.6 WRKY25 Transcription Factor
303(1)
6.14.7 WRKY48 Transcription Factor
303(1)
6.15 Manipulation of OsWRKY45 Transcription Factor-Dependent Priming Process Using Benzothiadiazole Compounds for Rice disease Management
304(4)
6.16 Manipulation of Priming of WRKY Transcription Factors Using BABA for Crop Disease Management
308(1)
6.17 Manipulation of WRKY Gene Expression Using Ergosterol for Disease Management
309(1)
6.18 Manipulation of MYB Transcription Factors for Disease Management
310(4)
6.18.1 Molecular Manipulation of MYB72 Transcription Factor Using Rhizobacteria to Trigger Priming and ISR for Disease Management
310(2)
6.18.2 Bioengineering MYB44 Transcription Factor for Management of Biotrophic/Hemibiotrophic Pathogens
312(1)
6.18.3 Bioengineering OsJAMyb for Rice Blast Disease Management
313(1)
6.19 Molecular Manipulation of MYC2 Transcription Factor Using Rhizobacteria to Trigger Priming and ISR for Disease Management
314(3)
6.20 Molecular Manipulation of bZIP Transcription Factors for Crop Disease Management
317(4)
6.20.1 Molecular Manipulation of TGA Class of bZIP Transcription Factors for Crop Disease Management
317(2)
6.20.2 Molecular Manipulation of RF2a and RF2b bZIP Transcription Factors for Rice Tungro Virus Disease Management
319(1)
6.20.3 Manipulation of Pepper bZIP Transcription Factor for Developing Disease-Resistant Plants
320(1)
6.21 Manipulation of EREBP Transcription Factors for Crop Disease Management
321(7)
6.21.1 EREBP Transcription Factor Family
321(1)
6.21.2 Pti5 Transcription Factor
321(1)
6.21.3 Pti4 Transcription Factor
322(1)
6.21.4 GbERF2 Transcription Factor
322(1)
6.21.5 NtERF5 Transcription Factor
323(1)
6.21.6 Tsil Transcription Factor
323(1)
6.21.7 OsBIERF3 Transcription Factor
324(1)
6.21.8 OsERF922 Transcription Factor
325(1)
6.21.9 CaPF1 Transcription Factor
326(1)
6.21.10 OPBP1 Transcription Factor
326(1)
6.21.11 HvRAF Transcription Factor
326(1)
6.21.12 ERF1 Transcription Factor
327(1)
6.21.13 OsEREBP1 Transcription Factor
327(1)
6.22 Manipulation of NAC Transcription Factors for Crop Disease Management
328(4)
6.22.1 NAC Transcription Factors in Plant Defense Responses
328(2)
6.22.2 Engineering NAC Transcription Factors for Disease Management
330(1)
6.22.3 Manipulation of NAC Transcription Factor Genes for Crop Disease Management
331(1)
6.22.4 NAC Transcription Factor Enhances ABA Biosynthesis and Promotes Disease Resistance
331(1)
6.23 Engineering NtWIF Transcription Factor Gene for Crop Disease Management
332(1)
6.24 Engineering AT-Hook Motif-Containing Transcription Factor Gene (CaATLI ) for Crop Disease Management
332(2)
References
334